Comment
doi: 10.1016/j.molcel.2019.04.015.
DNA Damage Meets Neurotrophin Signaling: A Delicate Balancing AKT to Maintain Virus Latency
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- PMID: 31051136
- DOI: 10.1016/j.molcel.2019.04.015
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DNA Damage Meets Neurotrophin Signaling: A Delicate Balancing AKT to Maintain Virus Latency
Mol Cell.
.
Free article
Abstract
In this issue of Molecular Cell, Hu et al. (2019) discover that coordinated regulation of AKT activity emanating from neurotrophic-factor stimulation and endogenous DNA damage maintains HSV latency. These studies provide novel insights into the role of AKT in integrating multiple signals to maintain neuronal homeostasis.
Copyright © 2019 Elsevier Inc. All rights reserved.
Comment on
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