Editorial: Cardiac Hypertrophy: From Compensation to Decompensation and Pharmacological Interventions
- PMID: 33981242
- PMCID: PMC8107717
- DOI: 10.3389/fphar.2021.665936
Editorial: Cardiac Hypertrophy: From Compensation to Decompensation and Pharmacological Interventions
Keywords: cardiac fibrosis; cardiac hypertrophy; cardiac remodeling; heart failure; heart function.
Conflict of interest statement
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
Similar articles
-
Nix-mediated apoptosis links myocardial fibrosis, cardiac remodeling, and hypertrophy decompensation.Circulation. 2008 Jan 22;117(3):396-404. doi: 10.1161/CIRCULATIONAHA.107.727073. Epub 2008 Jan 4. Circulation. 2008. PMID: 18178777 Free PMC article.
-
Osteopontin RNA aptamer can prevent and reverse pressure overload-induced heart failure.Cardiovasc Res. 2017 May 1;113(6):633-643. doi: 10.1093/cvr/cvx016. Cardiovasc Res. 2017. PMID: 28453726 Free PMC article.
-
Endothelial Forkhead Box Transcription Factor P1 Regulates Pathological Cardiac Remodeling Through Transforming Growth Factor-β1-Endothelin-1 Signal Pathway.Circulation. 2019 Aug 20;140(8):665-680. doi: 10.1161/CIRCULATIONAHA.119.039767. Epub 2019 Jun 10. Circulation. 2019. PMID: 31177814
-
Molecular switches under TGFβ signalling during progression from cardiac hypertrophy to heart failure.Br J Pharmacol. 2016 Jan;173(1):3-14. doi: 10.1111/bph.13344. Epub 2015 Nov 16. Br J Pharmacol. 2016. PMID: 26431212 Free PMC article. Review.
-
Mitochondria in cardiac hypertrophy and heart failure.J Mol Cell Cardiol. 2013 Feb;55:31-41. doi: 10.1016/j.yjmcc.2012.09.002. Epub 2012 Sep 13. J Mol Cell Cardiol. 2013. PMID: 22982369 Free PMC article. Review.
Cited by
-
Asporin, an extracellular matrix protein, is a beneficial regulator of cardiac remodeling.Matrix Biol. 2022 Jun;110:40-59. doi: 10.1016/j.matbio.2022.04.005. Epub 2022 Apr 22. Matrix Biol. 2022. PMID: 35470068 Free PMC article.
-
USP20 deletion promotes eccentric cardiac remodeling in response to pressure overload and increases mortality.Am J Physiol Heart Circ Physiol. 2024 Nov 1;327(5):H1257-H1271. doi: 10.1152/ajpheart.00329.2024. Epub 2024 Oct 4. Am J Physiol Heart Circ Physiol. 2024. PMID: 39365672
-
Transcription Factors Involved in the Development and Prognosis of Cardiac Remodeling.Front Pharmacol. 2022 Feb 2;13:828549. doi: 10.3389/fphar.2022.828549. eCollection 2022. Front Pharmacol. 2022. PMID: 35185581 Free PMC article. Review.
-
Postnatal Deletion of Bmal1 in Cardiomyocyte Promotes Pressure Overload Induced Cardiac Remodeling in Mice.J Am Heart Assoc. 2022 Jul 5;11(13):e025021. doi: 10.1161/JAHA.121.025021. Epub 2022 Jun 22. J Am Heart Assoc. 2022. PMID: 35730615 Free PMC article.
References
Publication types
LinkOut - more resources
Full Text Sources