Importance of the SDF-1:CXCR4 axis in myocardial repair
- PMID: 19461103
- PMCID: PMC2753196
- DOI: 10.1161/CIRCRESAHA.109.198929
Importance of the SDF-1:CXCR4 axis in myocardial repair
Figures
Comment on
-
Hypoxic preconditioning enhances the benefit of cardiac progenitor cell therapy for treatment of myocardial infarction by inducing CXCR4 expression.Circ Res. 2009 May 22;104(10):1209-16. doi: 10.1161/CIRCRESAHA.109.197723. Epub 2009 Apr 30. Circ Res. 2009. PMID: 19407239 Free PMC article.
Similar articles
-
Hypoxic preconditioning enhances the benefit of cardiac progenitor cell therapy for treatment of myocardial infarction by inducing CXCR4 expression.Circ Res. 2009 May 22;104(10):1209-16. doi: 10.1161/CIRCRESAHA.109.197723. Epub 2009 Apr 30. Circ Res. 2009. PMID: 19407239 Free PMC article.
-
Myocardial CXCR4 expression is required for mesenchymal stem cell mediated repair following acute myocardial infarction.Circulation. 2012 Jul 17;126(3):314-24. doi: 10.1161/CIRCULATIONAHA.111.082453. Epub 2012 Jun 9. Circulation. 2012. PMID: 22685115
-
Stromal cell-derived factor 1/CXCR4 signaling is critical for the recruitment of mesenchymal stem cells to the fracture site during skeletal repair in a mouse model.Arthritis Rheum. 2009 Mar;60(3):813-23. doi: 10.1002/art.24330. Arthritis Rheum. 2009. PMID: 19248097
-
SDF-1α as a therapeutic stem cell homing factor in myocardial infarction.Pharmacol Ther. 2011 Jan;129(1):97-108. doi: 10.1016/j.pharmthera.2010.09.011. Epub 2010 Oct 20. Pharmacol Ther. 2011. PMID: 20965212 Review.
-
Role of the SDF-1/CXCR4 system in myocardial infarction.Circ J. 2010 Mar;74(3):418-23. doi: 10.1253/circj.cj-09-1021. Epub 2010 Jan 30. Circ J. 2010. PMID: 20118565 Review.
Cited by
-
Aptamer-Based Proteomic Profiling Reveals Novel Candidate Biomarkers and Pathways in Cardiovascular Disease.Circulation. 2016 Jul 26;134(4):270-85. doi: 10.1161/CIRCULATIONAHA.116.021803. Circulation. 2016. PMID: 27444932 Free PMC article.
-
CXCR4+ Sorted Adipose-Derived Stem Cells Enhance Their Functional Benefits and Improve Cardiac Function after Myocardial Infarction.Stem Cells Int. 2022 Aug 23;2022:6714765. doi: 10.1155/2022/6714765. eCollection 2022. Stem Cells Int. 2022. PMID: 36051532 Free PMC article.
-
Integrative Genetic Approach Facilitates Precision Strategies for Acute Myocardial Infarction.Genes (Basel). 2023 Jun 26;14(7):1340. doi: 10.3390/genes14071340. Genes (Basel). 2023. PMID: 37510245 Free PMC article. Review.
-
Composite scaffold provides a cell delivery platform for cardiovascular repair.Proc Natl Acad Sci U S A. 2011 May 10;108(19):7974-9. doi: 10.1073/pnas.1104619108. Epub 2011 Apr 20. Proc Natl Acad Sci U S A. 2011. PMID: 21508321 Free PMC article.
-
Plasminogen regulates cardiac repair after myocardial infarction through its noncanonical function in stem cell homing to the infarcted heart.J Am Coll Cardiol. 2014 Jul 1;63(25 Pt A):2862-72. doi: 10.1016/j.jacc.2013.11.070. Epub 2014 Mar 26. J Am Coll Cardiol. 2014. PMID: 24681141 Free PMC article.
References
-
- Yamaguchi J, Kusano KF, Masuo O, Kawamoto A, Silver M, Murasawa S, Bosch-Marce M, Masuda H, Losordo DW, Isner JM, Asahara T. Stromal cell-derived factor-1 effects on ex vivo expanded endothelial progenitor cell recruitment for ischemic neovascularization. Circulation. 2003 March 11;107(9):1322–8. - PubMed
-
- Askari A, Unzek S, Popovic ZB, Goldman CK, Forudi F, kiedrowski M, Rovner A, Ellis SG, Thomas JD, DiCorleto PE, Topol EJ, Penn MS. Effect of stromal-cell-derived factor-1 on stem cell homing and tissue regeneration in ischemic cardiomyopathy. Lancet. 2003;362:697–703. - PubMed
-
- Deglurkar I, Mal N, Mills WR, Popovic ZB, McCarthy P, Blackstone EH, laurita KR, Penn MS. Mechanical and electrical effects of cell-based gene therapy for ischemic cardiomyopathy are independent. Hum Gene Ther. 2006 November;17(11):1144–51. - PubMed
-
- Zhang M, Mal N, kiedrowski M, Chacko M, Askari AT, Popovic ZB, Koc ON, Penn MS. SDF-1 expression by mesenchymal stem cells results in trophic support of cardiac myocytes after myocardial infarction. FASEB J. 2007 October;21(12):3197–207. - PubMed
Publication types
MeSH terms
Substances
Grants and funding
LinkOut - more resources
Full Text Sources
Other Literature Sources
Medical